T4 replacement therapy of hypophysectomized rats normalized LDL-c, but HDL-c and LLA were unaffected. During primary hypothyroidism, T4 treatment induced GH activity, increased LLA and reduced the HDL-c concentration. GH treatment of primary hypothyroid rats had a similar influence on the lipid levels and LLA as in hypophysectomized animals, although the lowering of HDL-c was.
In both the primary and secondary hypoproteins. In both the primary and secondary hypothyroid rats, total cholesterol (TC) was higher than in normal controls, due to an increase in low-density lipoprotein cholesterol (LDL-c) and to a lesser extent also in high-density lipoprotein cholesterol (HDL-c).
THIS SI BECAUSE THE PITUITARY GLAND HAS BEEN REMOVED. PITUITARY GLAND SECRETES TSH THAT CONTROLS PRODUCTION AND RELEASE OF THYROXINE, WITHOUT IT THE METABOLIC RATE IS EFFECTED AND IS SLOWER. front 4 ACTIVITY 2: WHAT EFFECT DID ADMINISTERING THYROXINE HAVE ON EACH OF THE RATS?
YOU CAN USE THIS TOOL TO DETERMINE THE CONCENTRATION OF GLUCOSE BY USINPECTROPHOTOMETER TO DETERMINE HOW MUCH GLUCOSE IS PRESENT ILOOD SAMPLE. front 18 ACTIVITY 7: WHICH PATIENT (S) HAD GLUCOSE READING (S) IN THE NORMAL RANGE?
Front 23 ACTIVITY 8: WHICH TWO PATIENT 'S HAVE HORMONE LEVELS CHARACTERISTIC OF CUSHING 'S SYNDROME? back 23 PATIENT 2 AND 5 HAVE HORMONE LEVELS THAT ARE SIMILAR TO THOSE OF CUSHING 'S SYNDROME.
Back 15 IT IMPROVED BONE DENSITY FROM -2.6 OSTEOPOROSIS TO -2.47 OSTEOPENIA ; ALTHOUGH IT WAS NOIG DIFFERENCE THE LEVELS IN CLASSIFICATION ARE DIFFERENT. front 16 ACTIVITY 5: HOW DID YOUR RESULTS COMPARE TO YOUR PREDICTIONS?
Front 24 ACTIVITY 8: PATIENT 2 IS BEING TREATED FOR RHEUMATOID ARTHRITIS WITH PREDNISONE. HOW DOES THIS CHANGE THE DIAGNOSIS? back 24 ITS NOT CUSHING 'S SYNDROME IF ITS IATROGENIC (PHYSICIAN INDUCED ).
Back 9 THE EFFECT OF PROPYLTHIOURACIL ON THE RATS MADE THE METABOLIC RATES REMAIN LOW. THIS IS BECAUSE PROPYLTHIOURACIL INHIBITS THYROXINE. SO THYROXINE IS NOT STIMULATED IN ANY RAT AND THE RATS THEREFORE CAN NOT MAINTAIN METABOLISM AND BODY HEAT MAKING NO CHANGE IN THE RATES.
THE PATIENT DOES NOT NORMALLY HAVE CUSHING 'S SYNDROME AND ITS PURPOSELY POSSIBLEMPORARY TREATMENT THAT WHEN IT STOPS BEING INDUCED MIGHT BE CORRECTED AGAIN. front 25 ACTIVITY 8: WHICH PATIENT WOULD MOST LIKELY BE DIAGNOSED WITH ADDISON 'S DISEASE?
Front 22 ACTIVITY 8: WHICH PATIENT WOULD MOST LIKELY BE DIAGNOSED WITH CUSHING 'S DISEASE? WHY? back 22 PATIENT 3; BECAUSE HYPERCORTISOLISM CAUSED BITUITARY TUMOR ALSO CAUSES LEVELS OF ACTH TO INCREASE, THIS PATIENT HAS BOTH THESE LEVELS HIGH SO IS MORE THEN LIKELY TO HAVE CUSHING 'S DISEASE.
See comment in PubMed Commons below. Author information 1Department of Internal Medicine III, University Hospital Dijkzigt, Rotterdam, The Netherlands. Abstract The aim of this study was to evaluate the effect of thyroxine (T4) and GH replacement therapy on serum lipoproteins in rats with primary and secondary hypothyroidism and to see whether recovery of GH activity.
I am glad I could help you out cause I wish I had someone to help me out when I took this course. I know Anatomy is super hard. back 5 I only ask that if you find these notecards helpful, you join Easy Notecards and create at least one notecard set to help others.
Front 10 ACTIVITY 4: DO YOU THINK THE DRUG PROPYLTHIOURACIL IS USED TO TREAT HYPOTHYROIDISM OR HYPERTHYROIDISM? EXPLAIN YOUR ANSWER. back 10. IT IS USED TO TREAT HYPERTHYROIDISM. SINCE IT INHIBITS THYROXINE IT CAN BE USED TO CONTROL THYROID PRODUCTION OF THYROXINE, AND FURTHER CONTROL HYPERTHYROIDISM.
Front 21 ACTIVITY 7: DESCRIBE THE DIAGNOSIS FOR PATIENT 3. back 21 TYPE 2 DIABETES MELLITUS - GLUCOSE REMAINS IN BLOODSTREAM UNABLE TO BE TAKEN UP BY THE BODY 'S CELLS TO SERVE AS THE PRIMARY FUEL FOR METABOLISM.
The thyroidectomized rat lacked thyroxine hormone. The normal rat lacked. 4. What was the effect of thyroxine on the thyroidectomized rat s metabolic rate?
Front 11 ACTIVITY 5: EXPLAIN WHY OVARIECTOMIZED RATS WERE USED IN THIS EXPERIMENT AND CORRELATE THIS TO THEIR BASELINCORE. back 11 THREE OVARIECTOMIZED RATS WERE USED BECASUE THEY ARE NO LONGER PRODUCING ESTROGEN DUE TO THE REMOVAL OF THEIR OVARIES.
Rat lacked TSH. The hypophysectomized rat lacked thyroxine. What was the effect of thyroxine on the thyroidectomized rat s metabolic rate? Thyroxine had.